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Asthma phenotyping and endotyping has emerged as a novel approach to asthma classification inspired by precision medicine which separates the clinical presentations of asthma, or asthma phenotypes, from their underlying causes, or asthma endotypes. The best-supported endotypic distinction is the type 2-high/type 2-low distinction.
During an asthma episode, inflamed airways react to environmental triggers such as smoke, dust, or pollen. The airways narrow and produce excess mucus, making it difficult to breathe. In essence, asthma is the result of an immune response in the bronchial airways. [3]
The most common cause of emphysema is smoking and smoking cessation is mandatory if this incurable disease is to be treated. Prevention of bronchoconstriction by this pathway is vital for people with emphysema and there are several anticholinergic medications that in combination with mucous thinning agents such as Guaifenesin cause significant ...
Asthma causes the bronchial tubes to swell and become inflamed upon encounter with certain triggers, making it difficult to breathe. Winter is riddled with triggers, making it a tough season for ...
"This causes recurrent episodes of coughing, wheezing, and chest tightness," says Dr. Laura Conrad, director of the Pediatric Asthma Center at the Children’s Hospital at Montefiore in New York.
It is hypothesized that C. pneumoniae is associated with asthma because C. pneumoniae has been found to cause ciliostasis in bronchial epithelial cells. [2] Meanwhile, sero-epidemiological data also provide evidences to support that C. pneumoniae plays a role in asthma by amplifying inflammation and inciting the disease process. [2]