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[45] [failed verification] UVC is the highest-energy, most-dangerous type of ultraviolet radiation, and causes adverse effects that can variously be mutagenic or carcinogenic. [46] Despite the importance of the sun to vitamin D synthesis, it is prudent to limit the exposure of skin to UV radiation from sunlight [47] and from tanning beds. [48]
Prolonged exposure to ultraviolet radiation from the sun can lead to melanoma and other skin malignancies. [4] Clear evidence establishes ultraviolet radiation, especially the non-ionizing medium wave UVB, as the cause of most non-melanoma skin cancers, which are the most common forms of cancer in the world. [4]
Phytophotodermatitis may be aggravated by the additional levels of ultraviolet light emitted by compact fluorescent light. Patients undergoing photodynamic therapy are at additional risk of adverse photosensitive reactions caused by compact fluorescent light. One cause of cataracts is exposure to ultraviolet light. Provided the level of UV ...
Ultraviolet radiation is thought to cause up to 95% of basal and squamous cell carcinomas, and between 70-95% of melanomas in people with fair skin. ... increased risk for disease,” she ...
Artificial UV light sources from tanning units and phototherapy treatment units can also trigger PLE. About three-quarters of patients acquire PLE after UV-A exposure only, one-tenth after UV-B exposure only, and the rest after a combination of UV-A and UV-B exposure. [6] People vary in the amount of sun exposure needed to trigger the rash. [15]
Excessive UV radiation is the leading cause of (primarily) non-malignant skin tumors, [1] [2] which in extreme cases can be life-threatening. Sunburn is an inflammatory response in the tissue triggered by direct DNA damage by UV radiation. When the cells' DNA is overly damaged by UV radiation, type I cell-death is triggered and the tissue is ...
The most important cause of AK formation is solar radiation, through a variety of mechanisms. Mutation of the p53 tumor suppressor gene, induced by UV radiation, has been identified as a crucial step in AK formation. [26] This tumor suppressor gene, located on chromosome 17p132, allows for cell cycle arrest when DNA or RNA is damaged.
Phytophotodermatitis, also known as berloque dermatitis, [1] [2] [3] margarita photodermatitis, [4] [5] lime disease [6] or lime phytodermatitis [6] is a cutaneous phototoxic inflammatory reaction resulting from contact with a light-sensitizing botanical agent (such as lime juice) followed by exposure to ultraviolet A (UV-A) light (from the sun, for instance).