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Azotemia has three classifications, depending on its causative origin: prerenal azotemia, renal azotemia, and postrenal azotemia. [2] Measurements of urea and creatinine (Cr) in the blood are used to assess renal function. For historical reasons, the lab test measuring urea is known as "blood urea nitrogen" (BUN) in the US. The BUN:Cr ratio is ...
prerenal disease: the physiologic response to a decrease in kidney perfusion is an increase in sodium reabsorption to control hyponatremia, often caused by volume depletion or decrease in effective circulating volume (e.g. low output heart failure). above 2% [citation needed] or 3% [2] acute tubular necrosis or other kidney damage (postrenal ...
The causes of acute kidney injury are commonly categorized into prerenal, intrinsic, and postrenal. Acute kidney injury occurs in up to 30% of patients following cardiac surgery. [ 22 ] Mortality increases by 60-80% in post-cardiopulmonary bypass patients who go on to require renal replacement therapy.
The above formula only applies for GFR calculation when it is equal to the clearance rate. The normal range of GFR, adjusted for body surface area , is 100–130 average 125 (mL/min)/(1.73 m 2 ) in men and 90–120 (mL/min)/(1.73 m 2 ) in women younger than the age of 40.
Prerenal (before the kidney) BUN reabsorption is increased. BUN is disproportionately elevated relative to creatinine in serum. This may be indicative of hypoperfusion of the kidneys due to heart failure or dehydration. [4] Gastrointestinal bleeding or increased dietary protein can also increase the ratio. [4] 12–20:1 40–110:1
Acute kidney injury (AKI), previously called acute renal failure (ARF), [12] [13] is a rapidly progressive loss of renal function, [14] generally characterized by oliguria (decreased urine production, quantified as less than 400 mL per day in adults, [15] less than 0.5 mL/kg/h in children or less than 1 mL/kg/h in infants); and fluid and ...
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