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Between 1% and 15% of people globally are affected by kidney stones at some point. [14] [15] In 2015, they caused about 16,000 deaths worldwide. [16] Some of the oxalate in urine is produced by the body. Calcium and oxalate in the diet play a part but are not the only factors that affect the formation of calcium oxalate stones.
Kidney stones can result from an underlying metabolic condition, such as distal renal tubular acidosis, [21] Dent's disease, [22] hyperparathyroidism, [23] primary hyperoxaluria, [24] or medullary sponge kidney. 3–20% of people who form kidney stones have medullary sponge kidney. [25] [26]
Human gallstones, all removed from one patient. Grid scale 1 mm. Calculi in the inner ear are called otoliths; Calculi in the urinary system are called urinary calculi and include kidney stones (also called renal calculi or nephroliths) and bladder stones (also called vesical calculi or cystoliths).
Struvite kidney stones are also known as triple phosphate stones (calcium magnesium ammonium phosphate), owing to the presence of carbonate apatite that precipitates to accompany struvite at high pH. [ 10 ] [ 11 ] [ 12 ] Struvite and carbonate apatite precipitate in alkaline urine , forming kidney stones.
Increased xylitol consumption can increase oxalate, calcium, and phosphate excretion to urine (termed oxaluria, calciuria, and phosphaturia, respectively). These are known risk factors for kidney stone disease, but despite that, xylitol has not been linked to kidney disease in humans. [38]