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Pulp necrosis is a clinical diagnostic category indicating the death of cells and tissues in the pulp chamber of a tooth with or without bacterial invasion. [1] It is often the result of many cases of dental trauma , caries and irreversible pulpitis .
It is a likely outcome of untreated dental caries (tooth decay), and in such cases it can be considered a sequela in the natural history of tooth decay, irreversible pulpitis and pulpal necrosis. Other causes can include occlusal trauma due to 'high spots' after restoration work, extrusion from the tooth of root filling material, or bacterial ...
The pulp is still alive, but the introduction of bacteria into the pulp will not allow the pulp to heal and it will ultimately result in necrosis, or death, of the pulp tissue. [ 11 ] Symptoms associated with irreversible pulpitis may include dull aching, pain from hot or cold (though cold may actually provide relief) lingering pain after ...
However, a lingering pain which continues despite the removal of the stimulus is indicative of irreversible pulpitis. No response — lack of response to sensitivity testing suggests that the nerve supply to the tooth has been diminished, as in the case of pulpal necrosis or in previously root treated canals.
Pulpitis is reversible when the pain is mild to moderate and lasts for a short time after a stimulus (for instance cold); or irreversible when the pain is severe, spontaneous, and lasts a long time after a stimulus. Left untreated, pulpitis may become irreversible, then progress to pulp necrosis (death of the pulp) and apical periodontitis ...
An abscess is a localized collection of pus which forms during an acute infection. The important difference between a periapical abscess and abscesses of the periodontium are that the latter do not arise from pulp necrosis. [12] Abscesses of the periodontium are categorized as gingival abscess, periodontal abscess and pericoronal abscess.
Initial stage: Epithelial cells from the rests of Malassez at the apex of the roots of a non-vital tooth (one where the nerve and blood supply in the tooth have degenerated and no longer exist) become stimulated due to the body's inflammatory response to bacterial endotoxins infecting the pulp or as a direct response to necrotic pulp tissue ...
Inflammation of the dental pulp, termed pulpitis, produces true hypersensitivity of the nerves in the dental pulp. [3] Pulpitis is classified as irreversible when pulpal inflammation will irreversibly progress to pulpal necrosis due to compression of the venous microcirculation and tissue ischemia, and reversible when the pulp is still capable ...