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Absence seizures are also known to occur to patients with porphyria and can be triggered by stress or other porphyrin-inducing factors. Childhood Absence Epilepsy. Childhood absence epilepsy (CAE) is a type of idiopathic epilepsy characterized by its non-convulsive, generalized nature and a genetic origin influenced by multiple factors [20]
It consists of repetitive and discontinuous episodes of eyelid myoclonia with mild absence, rather than continuous non- convulsive absence status epilepticus. Onset is typically in childhood with a peak at age 6–8 years (range 2–14 years). There is a twofold preponderance of girls. Prevalence and incidence is probably low.
Symptoms often manifest in difficulties with staring, mind blanking, absent-mindedness, mental confusion and maladaptive mind-wandering alongside delayed, sedentary or slow motor movements. [2] To scientists in the field, it has reached the threshold of evidence and recognition as a distinct syndrome.
Symptoms may include shaking, loss of consciousness, and loss of bladder control. [2] They may or may not be caused by either physiological or psychological conditions. [2] Physiological causes include fainting, sleep disorders, and heart arrhythmias. [2] [3] Psychological causes are known as psychogenic non-epileptic seizures. [3]
[5] [6] [7] These episodes of dyspnea can be recurrent and symptoms can range from mild to severe and prolonged in some cases. [5] Agitation and a sense of panic are not uncommon and can result in hospitalization. [6] Different subtypes of vocal cord dysfunction are characterized by additional symptoms.
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The symptoms and clinical manifestations of frontal lobe epilepsy can differ depending on which specific area of the frontal lobe is affected. [ 2 ] The onset of a seizure may be hard to detect since the frontal lobes contain and regulate many structures and functions about which relatively little is known. [ 3 ]