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  2. 16 Ways to Get Rid of Hives, According to Doctors - AOL

    www.aol.com/16-ways-rid-hives-according...

    Topical steroids, like hydrocortisone, also help relieve the itching that comes with hives, Dr. Kim says. These creams or gels, which you can purchase at your drugstore, also reduce inflammation ...

  3. Histamine intolerance - Wikipedia

    en.wikipedia.org/wiki/Histamine_intolerance

    Histamine intolerance is a presumed set of adverse reactions (such as flush, itching, rhinitis, etc.) to ingested histamine in food. The mainstream theory accepts that there may exist adverse reactions to ingested histamine, but does not recognize histamine intolerance as a separate medical condition that can be diagnosed. [1]

  4. Antihistamine - Wikipedia

    en.wikipedia.org/wiki/Antihistamine

    H 1-antihistamines work by binding to histamine H 1 receptors in mast cells, smooth muscle, and endothelium in the body as well as in the tuberomammillary nucleus in the brain. Antihistamines that target the histamine H 1-receptor are used to treat allergic reactions in the nose (e.g., itching, runny nose, and

  5. Histamine liberators - Wikipedia

    en.wikipedia.org/wiki/Histamine_liberators

    Histamine is an organic compound that primarily functions in service of the human body's immune responses as well as for the regulation of many physiological functions. [1] Since their discovery in 1910, [ 2 ] histamines have been known to trigger inflammatory responses such as itching as part of an immune response to foreign pathogens; for ...

  6. Itch - Wikipedia

    en.wikipedia.org/wiki/Itch

    Itch originating in the skin is known as pruritoceptive, and can be induced by a variety of stimuli, including mechanical, chemical, thermal, and electrical stimulation, or infection. The primary afferent neurons responsible for histamine-induced itch are unmyelinated C-fibres. [1] Nociceptors.

  7. Mast cell stabilizer - Wikipedia

    en.wikipedia.org/wiki/Mast_cell_stabilizer

    They block mast cell degranulation, stabilizing the cell and thereby preventing the release of histamine and related mediators. One suspected pharmacodynamic mechanism is the blocking of IgE-regulated calcium channels. Without intracellular calcium, the histamine vesicles cannot fuse to the cell membrane and degranulate. [1]