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The alpha-3 beta-4 nicotinic receptor, also known as the α3β4 receptor and the ganglion-type nicotinic receptor, [1] is a type of nicotinic acetylcholine receptor, consisting of α3 and β4 subunits.
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Beta-3 blocker will inactivate beta-3 receptor and stops the following action. [7] Beta 3 receptor is a G-protein coupled receptor, similar to beta-1 and beta-2 receptors. [7] The receptor is involved in G-as activation. [7] The receptor will also stimulate adenylyl cyclase. [7]
The adrenergic receptors or adrenoceptors are a class of G protein-coupled receptors that are targets of many catecholamines like norepinephrine (noradrenaline) and epinephrine (adrenaline) produced by the body, but also many medications like beta blockers, beta-2 (β 2) antagonists and alpha-2 (α 2) agonists, which are used to treat high ...
108015 Ensembl ENSG00000117971 ENSMUSG00000035200 UniProt P30926 Q8R493 RefSeq (mRNA) NM_000750 NM_001256567 NM_148944 RefSeq (protein) NP_000741 NP_001243496 NP_683746 Location (UCSC) Chr 15: 78.62 – 78.73 Mb Chr 9: 54.94 – 54.96 Mb PubMed search Wikidata View/Edit Human View/Edit Mouse Neuronal acetylcholine receptor subunit beta-4 is a protein that in humans is encoded by the CHRNB4 ...
The alpha-3 beta-2 nicotinic receptor, also known as the α3β2 receptor, is a type of nicotinic acetylcholine receptor, consisting of α3 and β2 subunits. It occurs alongside the more common α3β4 nicotinic receptor in autonomic ganglia , and as an facilitatory presynaptic autoreceptor at the neuromuscular junction (NMJ).
The alpha-4 beta-2 nicotinic receptor, also known as the α4β2 receptor, is a type of nicotinic acetylcholine receptor implicated in learning, [1] consisting of α4 and β2 subunits. [2] It is located in the brain , where activation yields post- and presynaptic excitation , [ 2 ] mainly by increased Na + and K + permeability.
Integrin alpha-3 is a protein that in humans is encoded by the ITGA3 gene. [5] [6] ITGA3 is an integrin alpha subunit.Together with beta-1 subunit, it makes up half of the α3β1 integrin duplex that plays a role in neural migration and corticogenesis, acted upon by such factors as netrin-1 and reelin.