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acute lymphoblastic leukemia [4] lymphoma [5] [6] post-splenectomy state [7] CARD11-related congenital B cell lymphocytosis (rare, also known as BENTA disease) [8] Causes of relative lymphocytosis include: age less than 2 years; acute viral infections; connective tissue diseases, [9] thyrotoxicosis, [10] Addison's disease, [11]
Some children with autoimmune lymphoproliferative disorders are heterozygous for a mutation in the gene that codes for the Fas receptor, which is located on the long arm of chromosome 10 at position 24.1, denoted 10q24.1. [3] This gene is member 6 of the TNF-receptor superfamily (TNFRSF6).
[1] [4] Lymphocytic esophagitis does not occur with high frequency in other gastrointestinal conditions where lymphocytosis is found in the mucosa, including lymphocytic colitis and lymphocytic gastritis; however, there is a disease association with coeliac disease wherein lymphocytic inflammation occurs in the small bowel after exposure to ...
Measured by flow cytometry: Normal values <2.5% total T cells; <1% of total lymphocytes in peripheral blood; Marked elevations >5% virtually pathognomonic for ALPS; Mild elevations also found in other autoimmune diseases; Thought to be cytotoxic T lymphocytes that have lost CD8 expression; Unknown if driver of disease or epiphenomenon
Epstein–Barr virus–associated lymphoproliferative diseases; F. ... Monoclonal B-cell lymphocytosis; ... This page was last edited on 2 April 2018, ...
Monoclonal B-cell lymphocytosis (MBL) is an asymptomatic condition in which individuals have increased blood levels of particular subtypes of monoclonal lymphocytes (i.e. an aberrant and potentially malignant group of lymphocytes produced by a single ancestral cell). This increase must persist for at least 3 months. [1]
Human herpes virus 8 (HHV8) is associated with four rare lymphoproliferative disorders: 1) a subset of diffuse large B cell lymphoma (DLBCL), b) large B-cell lymphoma arising in HHV8-associated multicentric Castleman's disease, c) primary effusion lymphoma, and 4) germinotropic lymphoproliferative disorder.
Secondary cold agglutinin syndrome occurs when autoantibodies bind to red blood cells, rendering them subject to attack by the complement system. [17] It is a result of an underlying condition potentially associated with either monoclonal cold-reacting autoantibodies or polyclonal cold-reacting autoantibodies [16] predominantly caused by infection or lymphoproliferative disorders. [16]