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Synesthesia (American English) or synaesthesia (British English) is a perceptual phenomenon in which stimulation of one sensory or cognitive pathway leads to ...
Mirror-touch synesthesia is found in approximately 1.6–2.5% of the general population. [6] Mirror-touch synesthetes have higher levels of affective and pain empathy than those without the condition, though cognitive empathy differs from person to person. [7]
Alternatively, synesthesia may arise through "disinhibited feedback" or a reduction in the amount of inhibition along feedback pathways (Grossenbacher & Lovelace 2001).It is well established that information not only travels from the primary sensory areas to association areas such as the parietal lobe or the limbic system, but also travels back in the opposite direction, from "higher order ...
Synesthesia is when input from one sense triggers the brain to associate it with another sensory experience. Dr. Hersh: Synesthesia occurs when more than one sense is experienced simultaneously ...
Pain synesthesia is a form of synesthesia that causes a person to experience pain when seeing pain empathetic eliciting stimuli. The most common group who report pain synesthesia are patients with phantom limb syndrome.
Wednesday Is Indigo Blue: Discovering the Brain of Synesthesia is a 2009 non-fiction book written by Richard Cytowic and David Eagleman documenting the current scientific understanding of synesthesia, a perceptual condition where an experience of one sense (such as sight) causes an automatic and involuntary experience in another sense (such as hearing). [1]
Synesthesia is a neurological condition in which two or more bodily senses are coupled. For example, in a form of synesthesia known as grapheme-color synesthesia, letters or numbers may be perceived as inherently colored. Historically, the most commonly described form of synesthesia (or synesthesia-like mappings) has been between sound and ...
When people with fibromyalgia are subjected to intense stimuli, they experience sensory overload in the form of pain. It is theorized that abnormal activity of the left dorsolateral prefrontal cortex and reduced production of or reception to serotonin are partially responsible for the sensation of pain in response to intense stimuli. [29]