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And the zombie cell-killing drugs didn’t seem to slow bone loss; it just improved bone creation rates. Perhaps most important, the treatment worked best on women who had high volumes of ...
Possible senolytic agents are under preliminary research, including some which are in early-stage human trials. [6] [7] [clarification needed] The majority of candidate senolytic compounds are repurposed anti-cancer molecules, such as the chemotherapeutic drug dasatinib and the experimental small molecule navitoclax.
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As we age, the body loses the ability to remove old, damaged cells that won’t grow or die, LeBrasseur. Over time, these “zombie cells” accumulate and increase inflammation.
Natural killer cells (NK cells) and macrophages play a major role in clearance of senescent cells. [65] Natural killer cells directly kill senescent cells, and produce cytokines which activate macrophages which remove senescent cells. [65] Senescent cells can be phagocytized by neutrophils as well as by macrophages. [66]
As they build up in your body, studies suggest, they promote aging and the conditions that come with it like osteoporosis and Alzheimer's disease. Basically the goal is to fight aging itself ...