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And the zombie cell-killing drugs didn’t seem to slow bone loss; it just improved bone creation rates. ... This research was the clinical trial to test a zombie cell-killer treatment in humans ...
The body removes most of them. But others linger like zombies. They aren't dead. But as the Mayo Clinic's Nathan LeBrasseur puts it, they can harm nearby cells like moldy fruit corrupting a fruit ...
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Possible senolytic agents are under preliminary research, including some which are in early-stage human trials. [6] [7] [clarification needed] The majority of candidate senolytic compounds are repurposed anti-cancer molecules, such as the chemotherapeutic drug dasatinib and the experimental small molecule navitoclax.
Natural killer cells directly kill senescent cells, and produce cytokines which activate macrophages which remove senescent cells. [65] Senescent cells can be phagocytized by neutrophils as well as by macrophages. [66] Senolytic drugs which induce apoptosis in senescent cells rely on phagocytic immune system cells to remove the apoptosed cells ...
For many years, neither "apoptosis" nor "programmed cell death" was a highly cited term. Two discoveries brought cell death from obscurity to a major field of research: identification of the first component of the cell death control and effector mechanisms, and linkage of abnormalities in cell death to human disease, in particular cancer.
The most notable components of the cell that are targets of cell damage are the DNA and the cell membrane. DNA damage: In human cells, both normal metabolic activities and environmental factors such as ultraviolet light and other radiations can cause DNA damage, resulting in as many as one million individual molecular lesions per cell per day. [5]
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