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Palmoplantar keratodermas are a heterogeneous group of skin disorders characterized by abnormal thickening (scleroderma) of the stratum corneum of the palms and soles. Autosomal recessive, dominant, X-linked, and acquired forms have all been described in medical literature. [1]: 505 [2]: 211 [3]
Quality of life can possibly can be decreased, therefore getting treatment is recommended. [6] Too much dry skin can be painful for some and cause discomfort. [ 8 ] There is limited data on the life expectancy of an affected person, but this disease alone does not reduce a person's lifespan.
Acrokeratoelastoidosis of Costa or Acrokeratoelastoidosis is a hereditary form of marginal keratoderma, and can be defined as a palmoplantar keratoderma. It is distinguished by tiny, firm pearly or warty papules on the sides of the hands and, occasionally, the feet.
Papillon–Lefèvre syndrome (PLS), also known as palmoplantar keratoderma with periodontitis, [1] [2] is an autosomal recessive [3] genetic disorder caused by a deficiency in cathepsin C. [ 4 ] [ 5 ]
Punctate palmoplantar keratoderma. Keratosis punctata palmaris et plantaris; Spiny keratoderma; Focal acral hyperkeratosis; Complex keratodermas Diffuse palmoplantar keratoderma. Erythrokeratodermia variabilis; Palmoplantar keratoderma of Sybert; Olmsted syndrome; Naegeli–Franceschetti–Jadassohn syndrome; Focal palmoplantar keratoderma
Haim–Munk syndrome (also known as "palmoplantar keratoderma with periodontitis and arachnodactyly, acro-osteolysis and Cochin Jewish syndrome" [1]) is a skin disease caused, like Papillon–Lefèvre syndrome, by a mutation in the cathepsin C gene. [1] One of its features is thick curved finger and toenails. [2]
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