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  2. Amyloidosis - Wikipedia

    en.wikipedia.org/wiki/Amyloidosis

    AL amyloidosis occurs in about 3–13 per million people per year and AA amyloidosis in about two per million people per year. [2] The usual age of onset of these two types is 55 to 60 years old. [2] Without treatment, life expectancy is between six months and four years. [2]

  3. AL amyloidosis - Wikipedia

    en.wikipedia.org/wiki/AL_amyloidosis

    AL amyloidosis is caused by the deposition of abnormal antibody free light chains. The abnormal light chains are produced by monoclonal plasma cells, and, although AL amyloidosis can occur without diagnosis of another disorder, it is often associated with other plasma cell disorders, such as multiple myeloma and Waldenström's macroglobulinemia. [6]

  4. Wild-type transthyretin amyloid - Wikipedia

    en.wikipedia.org/.../Wild-Type_Transthyretin_Amyloid

    Wild-type transthyretin amyloid (WTTA), also known as senile systemic amyloidosis (SSA), [1] is a disease that typically affects the heart and tendons of elderly people. It is caused by the accumulation of a wild-type (that is to say a normal ) protein called transthyretin .

  5. AA amyloidosis - Wikipedia

    en.wikipedia.org/wiki/AA_amyloidosis

    AA amyloidosis is a form of amyloidosis, a disease characterized by the abnormal deposition of fibers of insoluble protein in the extracellular space of various tissues and organs. In AA amyloidosis, the deposited protein is serum amyloid A protein (SAA), an acute-phase protein which is normally soluble and whose plasma concentration is highest ...

  6. Life expectancy gap between men and women in the US ... - AOL

    www.aol.com/life-expectancy-gap-between-men...

    Women in the United States can expect to live nearly six years longer than men, as disparities in deaths from Covid-19 and drug overdoses drive the life expectancy gap to the widest it’s been ...

  7. Familial amyloid polyneuropathy - Wikipedia

    en.wikipedia.org/wiki/Familial_amyloid_poly...

    In August 2018, the FDA approved patisiran, an siRNA-based treatment, at an expected cost of up to $450,000 per year. [16] In August 2021 six patients with hereditary ATTR amyloidosis with polyneuropathy were given doses of NTLA-2001, based on a CRISPR gene editing system. Researchers reported mild adverse events and decreases in serum ...