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Equine influenza (horse flu) is the disease caused by strains of influenza A that are enzootic in horse species. Equine influenza occurs globally, previously caused by two main strains of virus: equine-1 ( H7N7 ) and equine-2 ( H3N8 ). [ 1 ]
Also known as "horse flu" and "A1 influenza", the rapid outbreak was of the Influenza A virus strain of subtype H3N8. While the virus is highly contagious, it rarely kills adult horses but the performance of thoroughbred racing horses can be affected for several weeks. It can be fatal to young foals and debilitated horses.
Viruses that are shed for long periods of time after a horse gets better are much harder to control. Horses tend to be most infectious (i.e. shedding the most virus) in the first 24–48 hours after they develop a fever, but they can shed the virus for up to 7–10 days after their signs of illness disappear. [14]
This page was last edited on 14 September 2007, at 08:51 (UTC).; Text is available under the Creative Commons Attribution-ShareAlike 4.0 License; additional terms may apply.
Horse sickness may refer to: African horse sickness; Peruvian horse sickness This page was last edited on 6 August 2018, at 17:45 (UTC). Text is available under the ...
H7N7, however, has not been detected in horses since the late 1970s, [30] so it may have become extinct in horses. [20] H3N8 in equines spreads via aerosols and causes respiratory illness. [ 1 ] Equine H3N8 preferentially binds to α-2,3 sialic acids, so horses are usually considered dead-end hosts, but transmission to dogs and camels has ...
Following the destruction of Planet Southern Cross, the evil Anton flees and installs himself into a Zero unit to regain full mobility as the tesla coil-themed Anton Zero (アントンゼロ, Anton Zero). [104] [108] However, he is defeated by Shishi Red, Sasori Orange, Tenbin Gold, Oushi Black, Hebitsukai Silver, and Koguma Skyblue. Upon being ...
Horse flu; Human flu; Swine flu; Sources This page was last edited on 9 July 2022, at 22:53 (UTC). Text is available under the Creative Commons Attribution ...