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Manganese deficiency in humans results in several medical problems. [1] Manganese is a vital element of nutrition in very small quantities (adult male daily intake 2.3 milligrams). However, poisoning may occur when greater amounts are ingested.
Replenishing the deficit of dopamine with levodopa has been shown to initially improve extrapyramidal symptoms, [29] [30] [31] but the response to treatment goes down after 2 or 3 years, [32] with worsening condition of the same patients noted even after 10 years since last exposure to manganese. [33]
Manganese is an essential human dietary element. It is present as a coenzyme in several biological processes, which include macronutrient metabolism, bone formation, and free radical defense systems. It is a critical component in dozens of proteins and enzymes. [3] The human body contains about 12 mg of manganese, mostly in the bones.
Manganese is an essential human dietary element and is present as a coenzyme in several biological processes, which include macronutrient metabolism, bone formation, and free radical defense systems. Manganese is a critical component in dozens of proteins and enzymes. [8] The human body contains about 12 mg of manganese, mostly in the bones.
Niacin deficiency is a consequence of a diet low in both niacin and the amino acid tryptophan, a precursor for the vitamin. Low plasma tryptophan is a non-specific indicator, meaning it can have other causes. The signs and symptoms of niacin deficiency start to revert within days of oral supplementation with large amounts of the vitamin. [23] [24]
Ceruloplasmin (or caeruloplasmin) is a ferroxidase enzyme that in humans is encoded by the CP gene. [5] [6] [7] Ceruloplasmin is the major copper-carrying protein in the blood, and in addition plays a role in iron metabolism. It was first described in 1948. [8]