Search results
Results From The WOW.Com Content Network
Since pacemaker correction of the third-degree block requires full-time pacing of the ventricles, a potential side effect is pacemaker syndrome, and may necessitate the use of a biventricular pacemaker, which has an additional 3rd lead placed in a vein in the left ventricle, providing more coordinated pacing of both ventricles. [citation needed]
Emerging data also suggest that very frequent ventricular ectopy may be associated with cardiomyopathy through a mechanism thought to be similar to that of chronic right ventricular pacing associated cardiomyopathy. For patients with underlying chronic structural heart disease and complex ectopy, mortality is significantly increased. [3]
An ectopic pacemaker located in the atria is known as an atrial pacemaker and can cause the atrial contraction to be faster. [10] An ectopic pacemaker situated near the AV node and the septum is known as a junctional pacemaker. [11] The pacemaker that is operating in the ventricles is known as the ventricular. [12]
On ECG, there is no relationship between P waves and QRS complexes, meaning the P waves and QRS complexes are not in a 1:1 ratio. [7] Third-degree AV block is the most severe of the AV blocks. Persons with third-degree AV block need emergency treatment including but not limited to a pacemaker. [8]
AIVR appears similar to ventricular tachycardia with wide QRS complexes (QRS >0.12s) and a regular rhythm. It can most easily be distinguished from VT in that the rate is less than 120 and usually less than 100 bpm. There may or may not be AV dissociation depending on whether it is due to ventricular escape or AV block. [8]
Ectopic beat is a disturbance of the cardiac rhythm frequently related to the electrical conduction system of the heart, in which beats arise from fibers or group of fibers outside the region in the heart muscle ordinarily responsible for impulse formation (i.e., the sinoatrial node).
Intraventricular conduction delays (IVCD) are conduction disorders seen in intraventricular propagation of supraventricular impulses resulting in changes in the QRS complex duration or morphology, or both. IVCD can be caused by abnormalities in the structures of bundle of His, Purkinje fibers or ventricular myocardium.
This is measured from the initial deflection of the P wave to the beginning of the QRS complex. [3] In first-degree heart block, the AV node conducts the electrical activity more slowly. This is seen as a PR interval greater than 200 ms in length on the surface ECG. It is usually an incidental finding on a routine ECG. [4]