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In some cases of pulp necrosis there is a yellow, grey or brown crown discolouration. Dark coronal discoloration is believed to be an early sign of pulp degeneration. [16] Teeth with said discolouration need to be treated with special care and further investigations are required before pulp necrosis can be diagnosed. [11]
Pulp necrosis usually occurs either as ischaemic necrosis (infarction) caused by disruption to the blood supply at the apical foramen or as an infection-related liquefactive necrosis following dental trauma (2). Signs of pulpal necrosis include [42] Persistent grey colour to tooth that does not fade; Radiographic signs of periapical inflammation
Dental abscess; Other names: Dentoalveolar abscess, periapical abscess, tooth abscess, root abscess: A decayed, broken down tooth, which has undergone pulpal necrosis.A periapical abscess (i.e. around the apex of the tooth root) has then formed and pus is draining into the mouth via an intraoral sinus ().
b) Pulp Necrosis: The pulp tissue dies as a result of untreated pulpitis or direct trauma. Tooth discoloration; Infection: If bacteria penetrate the dentin and reach the pulp, it can lead to abscess formation or other periapical (around the root tip) infections. This can result in swelling, pain, and possible tooth loss if left untreated.
Inflammation therefore increases pressure in the pulp system, potentially compressing the blood vessels which supply the pulp. This may lead to ischemia (lack of oxygen) and necrosis (tissue death). Pulpitis is termed reversible when the inflamed pulp is capable of returning to a state of health, and irreversible when pulp necrosis is inevitable.
The pulp cannot recover from the insult and damage. For example, decay that has reached the pulp of the tooth introduces bacteria into the pulp. The pulp is still alive, but the introduction of bacteria into the pulp will not allow the pulp to heal and it will ultimately result in necrosis, or death, of the pulp tissue. [11]
Root resorption most commonly occurs due to inflammation caused by pulp necrosis, trauma, periodontal treatment, orthodontic tooth movement and tooth whitening. [3] Less common causes include pressure from malpositioned ectopic teeth, cysts , and tumors .
It is a likely outcome of untreated dental caries (tooth decay), and in such cases it can be considered a sequela in the natural history of tooth decay, irreversible pulpitis and pulpal necrosis. Other causes can include occlusal trauma due to 'high spots' after restoration work, extrusion from the tooth of root filling material, or bacterial ...