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Ventricular flutter is an arrhythmia, more specifically a tachycardia affecting the ventricles with a rate over 250-350 beats/min, and one of the most indiscernible. It is characterized on the ECG by a sinusoidal waveform without clear definition of the QRS and T waves.
They may be classified into narrow and wide complex based on the QRS complex. [10] Equal or less than 0.1s for narrow complex. [11] Presented in order of most to least common, they are: [10] Narrow complex Sinus tachycardia, which originates from the sino-atrial (SA) node, near the base of the superior vena cava; Atrial fibrillation; Atrial flutter
The types of SVT associated with TIC include atrial fibrillation, atrial flutter, incessant atrial tachycardia, permanent junctional reciprocating tachycardia, atrioventricular reciprocating tachycardia, and atrioventricular nodal reentry tachycardia. [1] Atrial fibrillation is the most common and well-studied etiology of TIC. [1] [5]
Absent P waves with "irregularly irregular" QRS complexes are the hallmark of atrial fibrillation. A "saw tooth" pattern with QRS complexes is the hallmark of atrial flutter. A sine wave pattern is the hallmark of ventricular flutter. Absent P waves with wide QRS complexes and a fast heart rate are ventricular tachycardia.
Atrial flutter was first identified as an independent medical condition in 1920 by the British physician Sir Thomas Lewis (1881–1945) and colleagues. [5] AFL is the second most common pathologic supraventricular tachycardia but occurs at a rate less than one-tenth of the most common supraventricular tachycardia (atrial fibrillation).
Ectopic beat is a disturbance of the cardiac rhythm frequently related to the electrical conduction system of the heart, in which beats arise from fibers or group of fibers outside the region in the heart muscle ordinarily responsible for impulse formation (i.e., the sinoatrial node).
Catheter ablation is a procedure that uses radio-frequency energy or other sources to terminate or modify a faulty electrical pathway from sections of the heart of those who are prone to developing cardiac arrhythmias such as atrial fibrillation, atrial flutter and Wolff-Parkinson-White syndrome.
Location of the pacemaker can also change its effect on the SA node and its rhythm. An ectopic pacemaker located in the atria is known as an atrial pacemaker and can cause the atrial contraction to be faster. [10] An ectopic pacemaker situated near the AV node and the septum is known as a junctional pacemaker. [11]