Search results
Results From The WOW.Com Content Network
Histopathology of placenta with increased syncytial knotting of chorionic villi, with two knots pointed out. The following characteristics of placentas have been said to be associated with placental insufficiency, however all of them occur in normal healthy placentas and full term healthy births, so none of them can be used to accurately diagnose placental insufficiency: [citation needed]
Amnioinfusion is a method in which isotonic fluid is instilled into the uterine cavity.. It was introduced in the 1960s as a means of terminating pregnancy and inducing labor in intrauterine death, but is currently used as a treatment in order to correct fetal heart rate changes caused by umbilical cord compression, indicated by variable decelerations seen on fetal heart rate monitoring.
On fetal heart tracing (a linear recording of the fetal heart rate) this would usually look like moderate to severe variable decelerations. [6] In overt cord prolapse, the cord can be seen or felt on the vulva or vagina. [1] The main issue with cord prolapse is that, once the cord is prolapsed, it is prone to compression by the foetus and the womb.
Presence of late decelerations with at least 50% of the contractions [9] Negative: No late or significant variable decelerations, [9] with at least 3 uterine contractions (lasting 40 seconds) in 10 minute period. [10] Equivocal—Suspicious: Presence of late decelerations with fewer than 50% of contractions or significant variable decelerations.
Late-life mortality deceleration is a well-established phenomenon in insects, [1] which often spend much of their lives in a constant hazard rate region, but it is much more controversial in mammals. [2] Rodent studies have found varying conclusions, with some finding short-term periods of mortality deceleration in mice, others not finding such ...
Fetal distress, also known as non-reassuring fetal status, is a condition during pregnancy or labor in which the fetus shows signs of inadequate oxygenation. [1] Due to its imprecision, the term "fetal distress" has fallen out of use in American obstetrics.
A 'gradual' deceleration has a time from onset to nadir of 30 seconds or more. Early decelerations begin and end at approximately the same time as contractions, and the low point of the fetal heart rate occurs at the peak of the contraction. [7] Late decelerations: a result of placental insufficiency, which can result in fetal distress ...
Intrauterine hypoxia can be attributed to maternal, placental, or fetal conditions. [12] Kingdom and Kaufmann classifies three categories for the origin of fetal hypoxia: 1) pre-placental (both mother and fetus are hypoxic), 2) utero-placental (mother is normal but placenta and fetus is hypoxic), 3) post-placental (only fetus is hypoxic).