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Mammalian cochlear hair cells are of two anatomically and functionally distinct types, known as outer, and inner hair cells. Damage to these hair cells results in decreased hearing sensitivity, and because the inner ear hair cells cannot regenerate, this damage is permanent. [4] Damage to hair cells can cause damage to the vestibular system and ...
Hensen's cells are important in ion metabolism and homeostasis regulation of both endolymph and perilymph, modulation of the hearing sensitivity, regulation and regeneration of the hair cells, and prevention of the cochlea damage. [6] The outer hair cells of the cochlea preprocess the signal by active movements, which can be elevated by ...
Proliferating supporting cells can acquire hair cell fate in mitotic division. The mouse's neonatal supporting cells proliferate after hair cell death and regenerate hair cells after damage. [26] The neonatal cochlea is resistant to hair cell damage caused by exposure to noise or drugs, which are toxic to the cochlea, or auditory nerve, in vivo ...
The drug is understood to damage multiple regions of the cochlea, causing the death of outer hair cells, as well as damage to the spiral ganglion neurons and cells of the stria vascularis. [27] Long-term retention of cisplatin in the cochlea may contribute to the drug's cochleotoxic potential. [ 28 ]
They are thought to damage the hair cells of the cochlea. Long-term exposure to these drugs may cause damage that progresses to the upper turn of the cochlea, impairing hearing or even causing deafness. [6] Glycopeptides, on the other hand, are rarely associated with ototoxicity.
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Oral mucositis is a common and often debilitating complication of cancer treatment. [3] Oral and gastrointestinal (GI) mucositis affects almost all patients undergoing high-dose chemotherapy and hematopoietic stem cell transplantation (HSCT), 80% of patients with cancers of the head and neck receiving radiotherapy, and a wide range of patients ...
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