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Lyme disease is the most common disease spread by ticks in the Northern Hemisphere. [21] [8] Infections are most common in the spring and early summer. [4] Lyme disease was diagnosed as a separate condition for the first time in 1975 in Lyme, Connecticut. It was originally mistaken for juvenile rheumatoid arthritis. [22]
Afzelius published his work 12 years later and speculated the rash came from the bite of an Ixodes tick, meningitic symptoms and signs in a number of cases and that both sexes were affected. This rash was known as erythema chronicum migrans, the skin rash found in early-stage Lyme disease. [18]
Allen Caruthers Steere is an American rheumatologist. He is a professor of rheumatology at Harvard University and previously at Tufts University and Yale University.Steere and his mentor, Stephen Malawista of Yale University, are credited with discovering and naming Lyme disease, and he has published almost 300 scholarly articles on Lyme disease during his more than 40 years of studies of this ...
About 30,000 cases of Lyme disease in the U.S. are reported to the Centers for Disease Control and Prevention by state and local health departments each year. However, the CDC says that many more ...
Lyme disease is caused by infected black-legged (or deer) ticks and symptoms of the disease may vary, depending on how long it takes to discover the signs. Show comments Advertisement
Anywhere from 30,000 up to 500,000 people develop Lyme disease from a tick bite each year, according to the C DC. For most, the infection is mild and easily treated with antibiotics.
Chronic Lyme disease is distinct from untreated late-stage Lyme disease, which can cause arthritis, peripheral neuropathy and/or encephalomyelitis. Chronic Lyme disease is also distinct from post-treatment Lyme disease syndrome (PTLDS) when symptoms linger after standard antibiotic treatments.
DRB1*04:01 is associated with multiple sclerosis, [12] rheumatoid arthritis, [13] type 1 diabetes, [14] [15] lyme disease induced arthritis. [16] HLA-DRB1*04:01 gene variant is found three times more often in asymptomatic carriers of SARS-CoV-2 than in patients with symptoms of COVID-19.
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