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Abdominal wall defects are a type of congenital defect that allows the stomach, the intestines, or other organs to protrude through an unusual opening that forms on the abdomen. [ 1 ] [ 2 ] During the development of the fetus, many unexpected changes occur inside the womb.
Defective ventral body wall formation yields a heart unprotected by the pericardium, sternum, or skin. Other organs may also have formed outside the skin, as well. Many cases of ectopia cordis have associated congenital heart defects, in which the heart has failed to properly form. [citation needed]
Abnormal folding of the body wall resulting in a ventral body wall defect through which the gut herniates [10] Failure to incorporate the yolk sac and related vitelline structures into the umbilical stalk [15] The first hypothesis does not explain why the mesoderm defect would occur in such a specific small area.
An omphalocele or omphalocoele, also known as an exomphalos, is a rare abdominal wall defect. [1] Beginning at the 6th week of development, rapid elongation of the gut and increased liver size reduces intra abdominal space, which pushes intestinal loops out of the abdominal cavity.
Abdominal wall defects: thoracoschisis and/or abdominoschisis; Limb defects; As a component of the abdominal wall defect, the umbilical cord is shortened or absent with the fetus being directly attached to the placenta, a key feature in its prenatal diagnosis by ultrasound. [3] Several systems have been proposed to classify LBWC cases ...
A Meckel's diverticulum, a true congenital diverticulum, is a slight bulge in the small intestine present at birth and a vestigial remnant of the vitelline duct.It is the most common malformation of the gastrointestinal tract and is present in approximately 2% of the population, [1] with males more frequently experiencing symptoms.
A Richter's hernia occurs when the antimesenteric wall of the intestine protrudes through a defect in the abdominal wall. This is distinct from other types of abdominal hernias in that only one intestinal wall protrudes through the defect, such that the lumen of the intestine is incompletely contained in the defect, while the rest remains in the peritoneal cavity.
Upon sectioning of the heart the atrioventricular endocardial cushions can be observed in the lumen of the atrial canal as two thickenings, one on its dorsal and another on its ventral wall. These thickenings will go on to fuse and remodel to eventually form the valves and septa of the mature adult heart.