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Hairy leukoplakia is a white patch on the side of the tongue with a corrugated or hairy appearance. It is caused by Epstein-Barr virus (EBV) and occurs usually in persons who are immunocompromised , especially those with human immunodeficiency virus infection/ acquired immunodeficiency syndrome (HIV/AIDS).
Oral hairy leukoplakia is a corrugated ("hairy") white lesion on the sides of the tongue caused by opportunistic infection with Epstein-Barr virus on a systemic background of immunodeficiency, almost always human immunodeficiency virus (HIV) infection. [15]
The tongue is only one of the 10 ways you can see disease written all over your face. There are a whole host of other reasons for bumps on the tongue. Bumps on the tongue come in many other varieties.
The tongue may traumatized by mechanical, thermal, electrical or chemical means. A common scenario is where the tongue is bitten accidentally whilst a local anesthetic inferior alveolar nerve block is wearing off. The tongue may develop scalloping on the lateral margins, sometimes termed crenated tongue.
a. Early lesions, blanching of the oral mucosa; b. Older lesions, vertical and circular palpable fibrous bands in and around the mouth or lips, resulting in a mottled, marble-like appearance of the buccal mucosa; Stage 3: Sequelae of oral submucous fibrosis a. Leukoplakia; b. Speech and hearing deficits
There is a similarity between this appearance and that of hairy leukoplakia, linea alba and leukoedema. [2] In people with human immunodeficiency virus , who are at higher risk of oral hairy leukoplakia, a tissue biopsy may be required to differentiate between this and frictional keratosis from cheek and tongue chewing.
A case study recently published in the New England Journal of Medicine is raising awareness about a condition being referred to as 'black hairy tongue.' Woman on antibiotics develops a 'black ...
Hairy tongue may be confused with hairy leukoplakia, however the latter usually occurs on the sides of the tongue and is associated with an opportunistic infection with Epstein–Barr virus on a background immunocompromise (almost always human immunodeficiency virus infection but rarely other conditions which suppress the immune system). [6]