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  2. Adenosine monophosphate deaminase deficiency type 1

    en.wikipedia.org/wiki/Adenosine_monophosphate...

    Adenosine monophosphate deaminase deficiency type 1 or AMPD1, is a human metabolic disorder in which the body consistently lacks the enzyme AMP deaminase, [1] in sufficient quantities. This may result in exercise intolerance, muscle pain and muscle cramping. The disease was formerly known as myoadenylate deaminase deficiency (MADD).

  3. Hypophosphatemia - Wikipedia

    en.wikipedia.org/wiki/Hypophosphatemia

    Hypophosphatemia is an electrolyte disorder in which there is a low level of phosphate in the blood. [1] Symptoms may include weakness, trouble breathing, and loss of appetite. [1]

  4. Mitochondrial disease - Wikipedia

    en.wikipedia.org/wiki/Mitochondrial_disease

    Mitochondrial disease is a group of disorders caused by mitochondrial dysfunction. Mitochondria are the organelles that generate energy for the cell and are found in every cell of the human body except red blood cells. They convert the energy of food molecules into the ATP that powers most cell functions.

  5. Mitochondrial myopathy - Wikipedia

    en.wikipedia.org/wiki/Mitochondrial_myopathy

    With ATP production deficient in mitochondria, there is an over-reliance on anaerobic glycolysis which leads to lactic acidosis either at rest or exercise-induced. [ 2 ] Primary mitochondrial myopathies are inherited, while secondary mitochondrial myopathies may be inherited (e.g. Duchenne's muscular dystrophy) [ 3 ] or environmental (e.g ...

  6. Metabolic myopathy - Wikipedia

    en.wikipedia.org/wiki/Metabolic_myopathy

    Some people with a metabolic myopathy never develop symptoms due to the body's ability to produce enough ATP through alternative pathways (e.g. the majority of those with AMP-deaminase deficiency are asymptomatic [1] [21]). H 2 O + ATP → H + + ADP + P i + energy → muscle contraction [22] ATP is needed for muscle contraction by two processes:

  7. MT-ATP6 - Wikipedia

    en.wikipedia.org/wiki/MT-ATP6

    Mitochondrial complex V deficiency is a shortage (deficiency) or loss of function in complex V of the electron transport chain that can cause a wide variety of signs and symptoms affecting many organs and systems of the body, particularly the nervous system and the heart. The disorder can be life-threatening in infancy or early childhood.

  8. Adenosine triphosphate - Wikipedia

    en.wikipedia.org/wiki/Adenosine_triphosphate

    The energy used by human cells in an adult requires the hydrolysis of 100 to 150 mol/L of ATP daily, which means a human will typically use their body weight worth of ATP over the course of the day. [30] Each equivalent of ATP is recycled 1000–1500 times during a single day (150 / 0.1 = 1500), [29] at approximately 9×10 20 molecules/s. [29]

  9. Adenine nucleotide translocator - Wikipedia

    en.wikipedia.org/wiki/Adenine_nucleotide_trans...

    Because a human typically exchanges the equivalent of their own mass of ATP on a daily basis, ADP/ATP translocase is an important transporter protein with major metabolic implications. [ 4 ] [ 8 ] ANT transports the free, i.e. deprotonated, non- Magnesium , non- Calcium bound forms of ADP and ATP , in a 1:1 ratio. [ 1 ]