Search results
Results From The WOW.Com Content Network
A rough-skinned newt underwater A rough-skinned newt at Brice Creek in Oregon. Throughout much of the newt's range, the common garter snake (Thamnophis sirtalis) has been observed to exhibit resistance to the tetrodotoxin produced in the newt's skin. While in principle the toxin binds to a tube-shaped protein that acts as a sodium channel in ...
The array of symptoms typically seen in carbamate poisoning include both muscarinic and nicotinic symptoms. [1] Muscarinic receptor effects include: Bradycardia, or reduced heart rate; Bronchospasm, or tightening of the bronchioles of the lungs; Miosis, or constriction of the pupils; Vomiting and abdominal pain; Diarrhea; Excessive sweating and ...
The Coastal Range newt produce a potent poison called tetrodotoxin from specialized glands in their skin. Ingestion of this poison by handling or eating them causes severe neurological symptoms and, in some instances, death from respiratory or cardiac failure. The toxin found in juveniles is several times more potent than that in adults.
Neurotoxic shellfish poisoning (NSP) is caused by the consumption of brevetoxins, which are marine toxins produced by the dinoflagellate Karenia brevis (among several others). These toxins can produce a series of gastrointestinal and neurological effects.
There are several causes of toxic optic neuropathy. [1] Among these are: ingestion of methanol (wood alcohol), ethylene glycol (automotive antifreeze), disulfiram (used to treat chronic alcoholism), halogenated hydroquinolones (amebicidal medications), ethambutol and isoniazid (tuberculosis treatment), and antibiotics such as linezolid and chloramphenicol as well as chloroquine and the related ...
Chronic solvent-induced encephalopathy (CSE) is a condition induced by long-term exposure to organic solvents, often—but not always—in the workplace, that lead to a wide variety of persisting sensorimotor polyneuropathies and neurobehavioral deficits even after solvent exposure has been removed.
Pyrrolizidine alkaloidosis can result in damage to the liver, kidneys, heart, brain, smooth muscles, lungs, DNA, lesions all over the body, and could be a potential cause of cancer. [ 1 ] [ 2 ] Pyrrolizidine alkaloidosis is known by many other names such as "Pictou Disease" in Canada [ 3 ] and "Winton Disease" in New Zealand. [ 4 ]
In milder cases, erethism and tremor regress slowly over a period of years following removal from exposure. Decreased nerve conduction velocity in mercury-exposed workers has been demonstrated. Long-term, low-level exposure has been found to be associated with less pronounced symptoms of erethism, characterized by fatigue, irritability, loss of ...