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Both HSV-1, and HSV-2 can be the cause of herpetic gingivostomatitis, [5] although HSV-1 is the source of infection in around 90% of cases. [6] Herpetic gingivostomatitis infections can present as acute or recurrent. Acute infection refers to the first invasion of the virus, and recurrent is when reactivation of the latent virus occurs. [7]
When inflammation of the gums and the mouth generally presents itself, sometimes the term gingivostomatitis is used, though this is also sometimes used as a synonym for herpetic gingivostomatitis. The term is derived from Greek στόμα (stoma) 'mouth' and -ῖτις (-itis) 'inflammation'.
The main symptom of oral infection is inflammation of the mucosa of the cheek and gums—known as acute herpetic gingivostomatitis—which occurs within 5–10 days of infection. Other symptoms may also develop, including headache, nausea, dizziness and painful ulcers —sometimes confused with canker sores —fever, and sore throat.
A diagnosis can be made from clinical signs and symptoms, and treatment consists of minimizing the discomfort of symptoms. [5] It can be differentiated from herpetic gingivostomatitis by the positioning of vesicles - in herpangina, they are typically found on the posterior oropharynx, as compared to gingivostomatitis where they are typically found on the anterior oropharynx and the mouth.
Necrotizing gingivitis (NG) is a common, non-contagious infection of the gums with sudden onset.The main features are painful, bleeding gums, and ulceration of interdental papillae (the sections of gum between adjacent teeth).
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If improperly treated necrotizing may become chronic and/or recurrent. In developed countries, necrotizing gingivitis occurs mostly in young adults with predisposing factors such as psychological stress, sleep deprivation, poor oral hygiene, smoking, immunosuppression and/or malnutrition. In developing countries, necrotizing gingivitis occurs ...
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