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A hiatal hernia or hiatus hernia [2] is a type of hernia in which abdominal organs (typically the stomach) slip through the diaphragm into the middle compartment of the chest. [1] [3] This may result in gastroesophageal reflux disease (GERD) or laryngopharyngeal reflux (LPR) with symptoms such as a taste of acid in the back of the mouth or heartburn.
Hiatal hernia [14] Cardiac bridge (Coronary occluding reflexes triggered by coronary reflexes) Enteric disease; Aneructonia, the loss of the ability to belch (continuous or intermittent) [citation needed] Bowel obstruction (Less common, this usually leads to intense pain in short time) Acute pancreatic necrosis [15] Eosinophilia
The causes are divided into benign or malignant. Benign Peptic ulcer disease; Infections, such as tuberculosis; and infiltrative diseases, such as amyloidosis.; A rare cause of gastric outlet obstruction is blockage with a gallstone, also termed "Bouveret syndrome" or "Bouveret's syndrome".
In one report 10% of 100 people investigated for iron deficiency anemia had a large hiatal hernia. [3] A 1967 review found that 20% of 1305 individuals having surgery for hiatal hernia were anemic. [4] Cameron in 1976 [5] compared 259 people with large hiatal hernias visible on chest x-ray with 259 controls without hernias. Present or past ...
Hiatus, or hiatal hernias often result in heartburn but may also cause chest pain or pain while eating. [3] Risk factors for the development of a hernia include smoking, chronic obstructive pulmonary disease, obesity, pregnancy, peritoneal dialysis, collagen vascular disease and previous open appendectomy, among others.
About one third of the cases are associated with a hiatal hernia. Treatment is surgical. The classic triad (Borchardt's Triad) of gastric volvulus, described by Borchardt in 1904, consists of severe epigastric pain, retching (due to sour taste in mouth) without vomiting, and inability to pass a nasogastric tube. It reportedly occurs in 70% of ...
[57] [59] A hiatal hernia repair is conducted, if necessary, with a posterior cruroplasty using a durable suture material. [59] This step is vital as it ensures the proper positioning of the gastroesophageal junction (GEJ) and reduces the risk of postoperative GERD by securing the stomach below the diaphragm, preventing potential acid reflux. [59]
The classic symptoms of GERD were first described in 1925, when Friedenwald and Feldman commented on heartburn and its possible relationship to a hiatal hernia. [14] In 1934 gastroenterologist Asher Winkelstein described reflux and attributed the symptoms to stomach acid. [15]