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Central nervous system fatigue is a key component in preventing peripheral muscle injury. [28] The brain has numerous receptors, such as osmoreceptors, to track dehydration, nutrition, and body temperature. With that information as well as peripheral muscle fatigue information, the brain can reduce the quantity of motor commands sent from the ...
The central component of fatigue is triggered by an increase of the level of serotonin in the central nervous system. [133] During motor activity, serotonin released in synapses that contact motor neurons promotes muscle contraction. [134] During high level of motor activity, the amount of serotonin released increases and a spillover occurs.
The existence of a central governor was suggested to explain fatigue after prolonged strenuous exercise in long-distance running and other endurance sports, but its ideas could also apply to other causes of exertion-induced fatigue. The existence of a central governor was proposed by Tim Noakes in 1997, but a similar idea was suggested in 1924 ...
Now it's widely known that the disease of the central nervous system, which often presents itself with visual problems, numbing, impaired balance, cognitive issues and fatigue, affects far more ...
[23]: 564 The classification of ME/CFS as a neurological disease is based on symptoms which indicate a central role of the nervous system. [24] Alternatively, on the basis of abnormalities in immune cells, ME/CFS is sometimes labelled a neuroimmune condition. [25]
The condition was explained as being a result of exhaustion of the central nervous system's energy reserves, which Beard attributed to modern civilization. Physicians in the Beard school of thought associated neurasthenia with the stresses of urbanization and with stress suffered as a result of the increasingly competitive business environment.
Synaptic fatigue, or short-term synaptic depression, is an activity-dependent form of short term synaptic plasticity that results in the temporary inability of neurons to fire and therefore transmit an input signal. It is thought to be a form of negative feedback in order to physiologically control particular forms of nervous system activity. [1]
The prevailing view is that fibromyalgia is a condition resulting from an amplification of pain by the central nervous system. [73] Substantial biological findings have backed up this notion, leading to development and adoption of the concept of nociplastic pain .