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Delayed sleep phase disorder (DSPD), more often known as delayed sleep phase syndrome and also as delayed sleep–wake phase disorder, is the delaying of a person's circadian rhythm (biological clock) compared to those of societal norms.
DSPD is a circadian rhythm sleep disorder, characterised by a mismatch between a person's internal biological clock and societal norms. [2] Chronotherapy uses the human phase response to light or melatonin. The American Academy of Sleep Medicine has recommended chronotherapy for the treatment of circadian rhythm and sleep disorders. [3]
Delayed sleep phase disorder (DSPD): Individuals who have been diagnosed with delayed sleep phase disorder have sleep–wake times that are delayed when compared to normal functioning individuals. People with DSPD typically have very long periods of sleep latency when they attempt to go to sleep during conventional sleeping times.
This sleep phase advancement can interfere with daily social and work schedules, and results in shortened sleep duration and excessive daytime sleepiness. [1] The timing of sleep and melatonin levels are regulated by the body's central circadian clock , which is located in the suprachiasmatic nucleus in the hypothalamus .
The related and more common delayed sleep phase disorder was not described until 1981. The first detailed study of non-24 in a blind subject was by Miles Le and his colleagues in 1977. The researchers reported on a 28-year-old male who had a 24.9-hour rhythm in sleep, plasma cortisol, and other parameters.
Among sighted individuals, Non-24 usually first appears in the teens or early twenties. As with delayed sleep phase disorder (DSPS or DSPD), in the absence of neurological damage due to trauma or stroke, cases almost never appear after the age of 30. [4] Non-24 affects more sighted males than sighted females. [4]