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Decreased exposure of the skin to sunlight is a common cause of vitamin D deficiency. [1] People with a darker skin pigment with increased amounts of melanin may have decreased production of vitamin D. [3] Melanin absorbs ultraviolet B radiation from the sun and reduces vitamin D production. [3] Sunscreen can also reduce vitamin D production. [3]
Exposure of skin to ultraviolet radiation from sunlight presents both positive and negative health effects. On the positive side, UV exposure enables the synthesis of vitamin D3, which is essential for bone health [1] and potentially plays a role in inhibiting certain cancers. [2][3] While vitamin D can also be obtained through dietary ...
A diet insufficient in vitamin D, combined with inadequate sunlight exposure, can lead to vitamin D deficiency, which is defined as a blood 25-hydroxyvitamin D or 25(OH)D level below 12 ng/mL (30 nmol/liter). Vitamin D insufficiency, on the other hand, is characterized by a blood 25(OH)D level between 12–20 ng/mL (30–50 nmol/liter).
Cholecalciferol, also known as vitamin D 3 or colecalciferol, is a type of vitamin D that is produced by the skin when exposed to UVB light; it is found in certain foods and can be taken as a dietary supplement. [3] Cholecalciferol is synthesised in the skin following sunlight exposure. [4]
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Even high-SPF sunscreens allow a small amount of UVB to reach the skin, sufficient for vitamin D synthesis. Additionally, brief, unprotected sun exposure can produce ample vitamin D, but this exposure also risks significant DNA damage and skin cancer. To avoid these risks, vitamin D can be obtained safely through diet and supplements.
But 5–15 minutes of casual sun exposure of hands, face and arms two to three times a week during the summer months is sufficient to keep your vitamin D levels high. Vitamin D can also be obtained from food and supplementation. [50] Excess sun exposure produces harmful effects, however. [49] Vitamin D promotes the creation of serotonin.
Excessive exposure to sunlight poses no risk in vitamin D toxicity through overproduction of vitamin D precursor, cholecalciferol, regulating vitamin D production. During ultraviolet exposure, the concentration of vitamin D precursors produced in the skin reaches an equilibrium, and any further vitamin D that is produced is degraded. [9]