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Cardiovascular physiology is the study of the cardiovascular system, specifically addressing the physiology of the heart ("cardio") and blood vessels ("vascular"). These subjects are sometimes addressed separately, under the names cardiac physiology and circulatory physiology .
In vertebrates, the circulatory system is a system of organs that includes the heart, blood vessels, and blood which is circulated throughout the body. [ 1 ] [ 2 ] It includes the cardiovascular system , or vascular system , that consists of the heart and blood vessels (from Greek kardia meaning heart , and Latin vascula meaning vessels ).
Heart attack. A heart attack results from a sudden plaque rupture and formation of a thrombus (blood clot) that completely blocks blood flow to a portion of the heart, leading to tissue death . CAD can also result in heart failure or arrhythmias. Heart failure is caused by chronic oxygen deprivation due to reduced blood flow, which weakens the ...
The heart is a muscular organ found in humans and other animals. This organ pumps blood through the blood vessels. [1] Heart and blood vessels together make the circulatory system. [2] The pumped blood carries oxygen and nutrients to the tissue, while carrying metabolic waste such as carbon dioxide to the lungs. [3]
Angiotensin II is a vasoconstrictor that will increase blood flow to the heart and subsequently the preload, ultimately increasing the cardiac output. Angiotensin II also causes an increase in the release of aldosterone from the adrenal glands. Aldosterone further increases the Na + and H 2 O reabsorption in the distal convoluted tubule of the ...
Vascular resistance is the resistance that must be overcome for blood to flow through the circulatory system.The resistance offered by the systemic circulation is known as the systemic vascular resistance or may sometimes be called by another term total peripheral resistance, while the resistance caused by the pulmonary circulation is known as the pulmonary vascular resistance.
This page was last edited on 31 December 2018, at 21:44 (UTC).; Text is available under the Creative Commons Attribution-ShareAlike 4.0 License; additional terms may apply.
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