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The documentation of the presence of petechiae on a victim can help police investigators prove the case. [11] Petechiae resulting from strangulation can be relatively tiny and light in color to very bright and pronounced. Petechiae may be seen on the face, in the whites of the eyes or on the inside of the eyelids.
Petechiae on the soft palate are mainly associated with streptococcal pharyngitis, [6] and as such it is an uncommon but highly specific finding. [ 7 ] 10 to 30 percent of palatal petechiae cases are estimated to be caused by suction, which can be habitual or secondary to fellatio .
Necrotizing sialometaplasia (NS) is a benign, ulcerative lesion, usually located towards the back of the hard palate. It is thought to be caused by ischemic necrosis (death of tissue due to lack of blood supply) of minor salivary glands in response to trauma. Often painless, the condition is self-limiting and should heal in 6–10 weeks.
flaccid paralysis, [1] such as soft palate weakness (examined by asking the patient to say aah). [1] muscle atrophy, [1] such as tongue atrophy with fasciculations. nasal speech lacking in modulation and difficulty with all consonants. drooling of saliva. [1] normal or absent jaw jerk. reduced or absent gag reflex. [1]
Forchheimer spots are a type of enanthem seen as tiny red spots on the soft palate in rubella, measles and scarlet fever. [1] They sometimes precede the skin rash of rubella. [1] The spots may be present in around 20% of people with rubella. [1] The sign is named after Frederick Forchheimer. [2]
The levator veli palatini (/ l ɪ ˈ v eɪ t ər ˈ v iː l aɪ ˌ p æ l ə ˈ t aɪ n aɪ /) is a muscle of the soft palate and pharynx. It is innervated by the vagus nerve (cranial nerve X) via its pharyngeal plexus. During swallowing, it contracts, elevating the soft palate to help prevent food from entering the nasopharynx.
Hypernasal speech is a disorder that causes abnormal resonance in a human's voice due to increased airflow through the nose during speech.It is caused by an open nasal cavity resulting from an incomplete closure of the soft palate and/or velopharyngeal sphincter (velopharyngeal insufficiency). [1]
Group III: Moderately advanced cases with an interincisal distance of 15–26 mm. Fibrotic bands are visible at the soft palate, and pterygomandibular raphe and anterior pillars of fauces are present. Group IVA: Trismus is severe, with an interincisal distance of less than 15 mm and extensive fibrosis of all the oral mucosa.