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Prednisolone has a relatively short half-life, ranging 2–4 hours. It also has a large therapeutic window, considering the dosage required to produce a therapeutic effect is a few times higher than what the body naturally produces. [14] Prednisolone is 70–90% plasma protein bound, it binds to proteins such as albumin. [14]
P4 [6] or P [3] 4-pregnene-3,20-dione 17α-Hydroxyprogesterone: 17-OHP4 [4] or 17OHP [7] or 17P4 [5] 17α-hydroxy-4-pregnene-3,20-dione Allopregnanolone: ALLO 5α-pregnan-3α-ol-20-one Androgens; Androstenedione: A4 [8] or AE [9] 4-androstene-3,17-dione 4-Hydroxyandrostenedione: 4-OH-A [3] 11β-Hydroxyandrostenedione: 11βOHA4 [10] or 11βOHΔ4 ...
Prednisone is a prodrug and must be converted to prednisolone by the liver before it becomes active. [6] [7] Prednisolone then binds to glucocorticoid receptors, activating them and triggering changes in gene expression. [4] Prednisone was patented in 1954 and approved for medical use in the United States in 1955.
Δ 7-Prednisolone, or 7-dehydroprednisolone, is a synthetic glucocorticoid corticosteroid which was never marketed. [1] [2] [3] References This page was last edited ...
Prednisolone acetate is acutely toxic with an LD50 of >240 mg/kg for a rat and 3500 mg/kg for a mouse. Effects may present delayed. Target organs include adrenal cortex, bones, and eyes. It is also a known teratogen. [3] Class B PPE should be worn when working with this chemical. Any contact with this chemical should be taken seriously and the ...