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Pleurisy, also known as pleuritis, is inflammation of the membranes that surround the lungs and line the chest cavity . [1] This can result in a sharp chest pain while breathing. [1] Occasionally the pain may be a constant dull ache. [5] Other symptoms may include shortness of breath, cough, fever, or weight loss, depending on the underlying cause.
Chest radiograph returns to normal in the majority of patients by 6 months. Patients should, of course, be advised to return sooner if symptoms redevelop. Long-term sequelae of pleural empyema are rare but include bronchopleural fistula formation, recurrent empyema and pleural thickening, which may lead to functional lung impairment needing ...
Pleural friction rubs are the squeaking or grating sounds of the pleural linings rubbing together [1] and can be described as the sound made by treading on fresh snow. They occur where the pleural layers are inflamed and have lost their lubrication. Pleural rubs are common in pneumonia, pulmonary embolism, and pleurisy (pleuritis).
Pleuritis or pleurisy is an inflammatory condition of pleurae. Due to the somatic innervation of the parietal pleura, pleural irritations, especially if from acute causes, often produce a sharp chest pain that is worse by breathing, known as pleuritic pain .
A parapneumonic effusion is a type of pleural effusion (accumulation of fluid in the pleural cavity) that arises as a result of a pneumonia, lung abscess, or bronchiectasis. [1]
Swimming induced pulmonary edema (SIPE), also known as immersion pulmonary edema, is a life threatening condition that occurs when fluids from the blood leak abnormally from the small vessels of the lung (pulmonary capillaries) into the airspaces (alveoli).
The symptoms tend to occur 2–3 weeks after myocardial infarction but can also be delayed a few months. It tends to subside in a few days, and very rarely leads to pericardial tamponade . [ 8 ] Elevated ESR is an objective but nonspecific laboratory finding.
Hydrothorax is the synonym of pleural effusion in which fluid accumulates in the pleural cavity.This condition is most likely to develop secondary to congestive heart failure, following an increase in hydrostatic pressure within the lungs.