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The body of the superior oblique muscle is located behind the eyeball, but the tendon (redirected by the trochlea) approaches the eyeball from the front. The tendon attaches to the top (superior aspect) of the eyeball at an angle of 51 degrees concerning the primary position of the eye (looking straight forward). Therefore, the force of the ...
The superior oblique muscle loops through a pulley-like structure (the trochlea of superior oblique) and inserts into the sclera on the posterotemporal surface of the eyeball. It is the pulley system that gives superior oblique its actions, causing depression of the eyeball despite being inserted on the superior surface. Superior oblique nerve
An injury to the trochlear nucleus in the brainstem will result in an contralateral superior oblique muscle palsy, whereas an injury to the trochlear nerve (after it has emerged from the brainstem) results in an ipsilateral superior oblique muscle palsy. The superior oblique muscle which the trochlear nerve innervates ends in a tendon that ...
The superior oblique portion arises from the anterior tubercles of the transverse processes of the third, fourth, and fifth cervical vertebrae and, ascending obliquely with a medial inclination, is inserted by a narrow tendon into the tubercle on the anterior arch of the atlas.
Trochleitis is inflammation of the superior oblique tendon trochlea apparatus characterized by localized swelling, tenderness, and severe pain. This condition is an uncommon but treatable cause of periorbital pain. The trochlea is a ring-like apparatus of cartilage through which passes the tendon of the superior oblique
The superior oblique muscle originates at the back of the orbit (a little closer to the medial rectus, though medial to it), getting rounder as it [5] courses forward to a rigid, cartilaginous pulley, called the trochlea, on the upper, nasal wall of the orbit. The muscle becomes tendinous about 10mm before it passes through the pulley, turning ...
Brown syndrome is caused by a malfunction of the superior oblique muscle, causing the eye to have difficulty moving up, particularly during adduction (when eye turns towards the nose). Harold W. Brown first described the disorder in 1950 and initially named it the "superior oblique tendon sheath syndrome". [1]
Tenon's capsule is perforated by the tendons of the ocular muscles and is reflected backward on each as a tubular sheath. The sheath of the obliquus superior is carried as far as the fibrous pulley of that muscle, and that on the obliquus inferior reaches as far as the floor of the orbit, to which it gives off a slip.