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Humans are the main indoor source of carbon dioxide (CO 2) in most buildings. Indoor CO 2 levels are an indicator of the adequacy of outdoor air ventilation relative to indoor occupant density and metabolic activity. Indoor CO 2 levels above 500 ppm can lead to higher blood pressure and heart rate, and increased peripheral blood circulation. [85]
Indoor PM 2.5 exposure levels have been consistently reported to be in the range of hundreds to thousands of micrograms per cubic meter (μg/m 3). Similarly, CO exposure levels have been measured to be as high as hundreds to greater than 1000 milligrams per cubic meter (mg/m 3 ).
For this model the standard provides a graph of acceptable indoor temperature limits at prevailing mean outdoor temperatures (a mean of the daily mean outdoor temperatures of the previous 7–30 days). An accompanying table lists provisions for higher operative temperatures at air speeds above 0.3 m/s (59 ft/min) and up to 1.2 m/s (240 ft/min).
Ventilation guidelines are based on the minimum ventilation rate required to maintain acceptable levels of effluents. Carbon dioxide is used as a reference point, as it is the gas of highest emission at a relatively constant value of 0.005 L/s. The mass balance equation is: Q = G/(C i − C a) Q = ventilation rate (L/s) G = CO 2 generation rate
Another set of standards, for indoor air in employment settings, is administered by the U.S. Occupational Safety and Health Administration. [7] A distinction may be made between mandatory and aspirational air quality standards. For example, U.S. state governments must work toward achieving NAAQS, but are not forced to meet them.
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NO 2 is the oxide measured and used as the indicator for the entire NO x family as it is of the most concern due to its quick formation and contribution to the formation of harmful ground level ozone. [18] In 1971 the primary and secondary NAAQS for NO 2 were both set at an annual average of 0.053 ppm. The EPA reviewed this NAAQS in 1985 and ...
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