Search results
Results From The WOW.Com Content Network
Right ventricular hypertrophy (RVH) is a condition defined by an abnormal enlargement of the cardiac muscle surrounding the right ventricle. The right ventricle is one of the four chambers of the heart.
Specifically, an increase in Q wave size, abnormalities in the P wave, as well as giant inverted T waves, are indicative of significant concentric hypertrophy. [13] Specific changes in repolarization and depolarization events are indicative of different underlying causes of hypertrophy and can assist in the appropriate management of the condition.
The electrical axis of the heart is the net direction in which the wave of depolarization travels. It is measured using an electrocardiogram (ECG).Normally, this begins at the sinoatrial node (SA node); from here the wave of depolarisation travels down to the apex of the heart.
The pathophysiology of pulmonary heart disease (cor pulmonale) has always indicated that an increase in right ventricular afterload causes RV failure (pulmonary vasoconstriction, anatomic disruption/pulmonary vascular bed and increased blood viscosity are usually involved [1]), however most of the time, the right ventricle adjusts to an overload in chronic pressure.
Repolarization of the ventricle happens in the opposite direction of depolarization and is negative current, signifying the relaxation of the cardiac muscle of the ventricles. But this negative flow causes a positive T wave; although the cell becomes more negatively charged, the net effect is in the positive direction, and the ECG reports this ...
Current standard ECG markers of repolarization abnormalities include ST depression, T wave inversion and QT prolongation. Many studies have investigated the prognostic strength of the SA for cardiac morbidity and mortality compared to these and other ECG parameters.
The clinician must therefore be well versed in recognizing the so-called ECG mimics of acute myocardial infarction, which include left ventricular hypertrophy, left bundle branch block, paced rhythm, early repolarization, pericarditis, hyperkalemia, and ventricular aneurysm. [7] [8] [9] Localisation of the occlusion in the ECG showing STEMI changes
The cardiac action potential has five phases. I to1 is active during phase 1, causing a fast repolarization of the action potential. The cardiac transient outward potassium current (referred to as I to1 or I to [1]) is one of the ion currents across the cell membrane of heart muscle cells.