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Lithium is readily absorbed from the gastrointestinal tract. [5] It is distributed to the body with higher levels in the kidney, thyroid, and bone as compared to other tissues. Since lithium is almost exclusively excreted by the kidneys, people with preexisting chronic kidney disease are at high risk of developing lithium intoxication. [13]
Continued use of lithium can lead to more serious kidney damage in an aggravated form of diabetes insipidus. [90] [91] In rare cases, some forms of lithium-caused kidney damage may be progressive and lead to end-stage kidney failure with a reported incidence of 0.2% to 0.7%. [92]
The most obvious cause is a kidney or systemic disorder, including amyloidosis, [2] polycystic kidney disease, [3] electrolyte imbalance, [4] [5] or some other kidney defect. [2] The major causes of acquired nephrogenic diabetes insipidus that produce clinical symptoms (e.g., polyuria) in the adult are lithium toxicity and high blood calcium.
Nephrotoxicity is toxicity in the kidneys. It is a poisonous effect of some substances, both toxic chemicals and medications, on kidney function. [1] There are various forms, [2] and some drugs may affect kidney function in more than one way. Nephrotoxins are substances displaying nephrotoxicity.
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High dietary protein intake is not associated with kidney disease in healthy people. [322] While significantly increased protein intake in the short-term is associated with changes in renal function, there is no evidence to suggest this effect persists in the long-term and results in kidney damage or disease. [323]
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The quarantine spared the lives of approximately 8,000 men, women and children from being deported to concentration camps. Lazowski kept his activities a secret, not even telling his wife.
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