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The most common and simple treatment is the construction of a specially made acrylic prosthesis that covers the biting surfaces of the teeth and protects the cheek, tongue, and labial mucosa (an occlusal splint). This is either employed in the short term as a habit-breaking intention or more permanently (e.g., wearing the prosthesis each night ...
The aetiology of dental abrasion can be due to a single stimulus or, as in most cases, multi-factorial. [6] The most common cause of dental abrasion is the combination of mechanical and chemical wear. Tooth brushing is the most common cause of dental abrasion, which is found to develop along the gingival margin, due to vigorous brushing in this ...
Abrasion is loss of tooth substance caused by physical means other than teeth. [10] The term is derived from the Latin verb abrasum, which means ‘to scrape off’. [2] It tends to present as rounded ditching around the cervical margins of teeth, commonly described as ‘shallow’, concave or wedge shaped notches. [1]
Transient lingual papillitis is generally diagnosed based on patient presentation, meaning where it is located in the mouth and how big the bump is. [8] The visual presentation can also accompany various signs and symptoms such as difficulty eating, having a "strawberry tongue", increased saliva production, and a burning or tingling sensation. [9]
Lymphangiomas are far less common in the oral mucosa, usually appearing on the tongue, less commonly the lip at birth or in infancy. They are colourless to pale pink and may be nodular projections or resemble "frog spawn" domes. They can cause macroglossia (enlargement of the tongue).
He's the ShamWow guy. You know: "Vince," that guy who's stealing Billy Mays' infomercial crown with a smoother spiel and a raffish, aging-frat-boy charm. He's currently hawking his super-absorbent ...
In 1890, W.D. Miller, considered the father of oral microbiology, was the first to associate pulpal disease with the presence of bacteria. [11] This was confirmed by Kakehashi, who, in 1965, proved that bacteria were the cause of pulpal and periradicular disease in studies using animal models; pulpal exposures were initiated in both normal and germ-free rats, and while no pathologic changes ...
The condition may affect only part of the tongue, with a predilection for the tip and the sides of the tongue, [4] or the entire dorsal surface at any one time. The condition goes through periods of remission and relapse. Loss of the white peripheral zone is thought to signify periods of mucosal healing. [9]