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The cerebellum is also responsible for refining crude motor output from the primary motor cortex. When this refinement is missing, symptoms such as unsteadiness and ataxia [8] will present. A potential cause of chronic alcoholic cerebellar dysfunction is an alteration of GABA-A receptor.
Some people may experience frequent falls and gait unsteadiness due to ataxia. This ataxia may be caused by cerebellar degeneration, sensory ataxia, or distal muscle weakness. [4] Over time, alcoholic polyneuropathy may also cause difficulty swallowing , speech impairment , muscle spasms, and muscle atrophy. [5]
Cerebellar ataxia is a form of ataxia originating in the cerebellum. [1] Non-progressive congenital ataxia (NPCA) is a classical presentation of cerebral ataxias. Cerebellar ataxia can occur as a result of many diseases and may present with symptoms of an inability to coordinate balance, gait, extremity and eye movements. [ 2 ]
As a stand-alone condition, "ataxia is a degenerative disease of the nervous system," explains Andrew Rosen, chief executive officer of the National Ataxia Foundation. There are many types ...
Wernicke-Korsakoff syndrome (WKS) is the combined presence of Wernicke encephalopathy (WE) and alcoholic Korsakoff syndrome (AKS [clarification needed]).Due to the close relationship between these two disorders, people with either are usually diagnosed with WKS as a single syndrome.
Ataxia (from Greek α- [a negative prefix] + -τάξις [order] = "lack of order") is a neurological sign consisting of lack of voluntary coordination of muscle movements that can include gait abnormality, speech changes, and abnormalities in eye movements, that indicates dysfunction of parts of the nervous system that coordinate movement, such as the cerebellum.
[21] [22] Nutritional deficiency is a non-inherited condition that lead to impaired absorption or utilisation of the vitamin thiamine (B-1) by the body, thus causing temporary or permanent damage to cerebellar cells. [2] Alcoholic degeneration of cerebellar cells is the most common trigger of spinocerebellar ataxia. [23]
It is associated with cerebellar ataxia, [1] [2] when the cerebellum has been damaged and does not function to its fullest ability. Lesions to the cerebellum can cause dyssynergia, dysmetria, dysdiadochokinesia, dysarthria, and ataxia of stance and gait. [3] Dyschronometria can result from autosomal dominant cerebellar ataxia (ADCA). [4]