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CT and MRI are most commonly used to observe the brain for cerebral atrophy. A CT scan takes cross sectional images of the brain using X-rays, while an MRI uses a magnetic field. With both measures, multiple images can be compared to see if there is a loss in brain volume over time. [20]
The term "leukoaraiosis" was coined in 1986 [6] [7] by Hachinski, Potter, and Merskey as a descriptive term for rarefaction ("araiosis") of the white matter, showing up as decreased density on CT and increased signal intensity on T2/FLAIR sequences (white matter hyperintensities) performed as part of MRI brain scans.
Accumulation of extracellular fluid increases brain volume and then intracranial pressure causing the symptoms of cerebral edema. [1] There are several clinical conditions in which vasogenic edema is present: CNS tumors, like glioblastoma and meningioma [1] [21] Infections like meningitis, abscess, and encephalitis [1] [21]
MRI scans showing hyperintensities. A hyperintensity or T2 hyperintensity is an area of high intensity on types of magnetic resonance imaging (MRI) scans of the brain of a human or of another mammal that reflect lesions produced largely by demyelination and axonal loss.
For a small object (e.g., a voxel) or an object of size comparable to the spatial resolution of the imaging system, the observed activity is the sum of activity due to partial volume loss plus spillover from adjacent regions. The method to correct for the partial volume effect is referred to as partial volume correction (see [3] [4] [5] [6]).
The first study of the human brain at 3.0 T was published in 1994, [13] and in 1998 at 8 T. [14] Studies of the human brain have been performed at 9.4 T (2006) [15] and up to 10.5 T (2019). [16] Paul Lauterbur and Sir Peter Mansfield were awarded the 2003 Nobel Prize in Physiology or Medicine for their discoveries concerning MRI.
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Fogging phenomenon in computerized tomography (CT) scanning of the head is vanishing signs of an infarct on the serial CT imaging in a patient with a recent stroke. [1] It is a reversal of the hypodensity on the CT after an acute ischemic stroke.