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Manganese superoxide dismutase (MnSOD) is the principal antioxidant in mitochondria. Several enzymes activated by manganese contribute to the metabolism of carbohydrates, amino acids, and cholesterol. [2] A deficiency of manganese causes skeletal deformation in animals and inhibits the production of collagen in wound healing. [3]
Manganese is an essential human dietary element. It is present as a coenzyme in several biological processes, which include macronutrient metabolism, bone formation, and free radical defense systems. It is a critical component in dozens of proteins and enzymes. [3] The human body contains about 12 mg of manganese, mostly in the bones.
Acrodermatitis enteropathica is an autosomal recessive metabolic disorder affecting the uptake of zinc through the inner lining of the bowel, the mucous membrane.It is characterized by inflammation of the skin around bodily openings (periorificial) and the tips of fingers and toes (acral), hair loss (), and diarrhea.
Menkes disease (MNK), also known as Menkes syndrome, [1] [2] is an X-linked recessive disorder caused by mutations in genes coding for the copper-transport protein ATP7A, [3] leading to copper deficiency. [4] [5] Characteristic findings include kinky hair, growth failure, and nervous system deterioration. Like all X-linked recessive conditions ...
This gene is a mitochondrial member of the iron/manganese superoxide dismutase family. [5] [7] It encodes a mitochondrial matrix protein that forms a homotetramer and binds one manganese ion per subunit. [5] [6] The manganese site forms a trigonal bipyramidal geometry with four ligands from the protein and a fifth solvent ligand. This solvent ...
Manganese deficiency may refer to: Manganese deficiency (medicine) Manganese deficiency (plant) This page was last edited on 29 December 2019, at 08:32 (UTC). ...
Mineral deficiency is a lack of the dietary minerals, the micronutrients that are needed for an organism's proper health. [1] The cause may be a poor diet , impaired uptake of the minerals that are consumed, or a dysfunction in the organism's use of the mineral after it is absorbed.
Emaciation manifests physically as thin limbs, pronounced and protruding bones, sunken eyes, dry skin, thinning hair, a bloated stomach, and a dry or coated tongue in humans. Emaciation is often accompanied by halitosis, hyponatremia, hypokalemia, anemia, improper function of lymph and the lymphatic system, and pleurisy and edema. [2]