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Caffeine dependence can cause a host of physiological effects if caffeine consumption is not maintained. Commonly known caffeine withdrawal symptoms include headaches, fatigue, loss of focus, lack of motivation, mood swings, nausea, insomnia, dizziness, cardiac issues, hypertension, anxiety, and backache and joint pain; these can range in severity from mild to severe. [18]
Dr. Rohit Vuppuluri, an interventional cardiologist, tells Yahoo Life that caffeine is safe to use in moderation — less than 400 mg per day, according to the Food and Drug Administration (FDA ...
Caffeine is considered one of the most widely consumed drugs around the world. Around 80% of the world population consumes caffeine in one form or another. [ 2 ] It is found in coffee , tea , caffeinated alcoholic drinks , cocoa , chocolate , soft drinks , especially cola , and is an important component of energy drinks and other dietary ...
Caffeine withdrawal = fatigue, nausea, muscle pain and wicked headaches. Jeffrey Goldberger, a University of Miami cardiologist and ... For caffeine drinkers having surgery, withdrawal might be ...
"Caffeine is a drug, and like all drugs there are benefits and risks associated with its consumption." For those who have questions or concerns about the amount or timing of coffee consumption, ...
The US Food and Drug Administration (FDA) considers safe beverages containing less than 0.02% caffeine; [291] but caffeine powder, which is sold as a dietary supplement, is unregulated. [292] It is a regulatory requirement that the label of most prepackaged foods must declare a list of ingredients, including food additives such as caffeine, in ...
Medications are used to reverse the symptoms of extrapyramidal side effects caused by antipsychotics or other drugs, by either directly or indirectly increasing dopaminergic neurotransmission. The treatment varies by the type of the EPS, but may involve anticholinergic agents such as procyclidine, benztropine, diphenhydramine, and trihexyphenidyl.
The term was first used by Czech neuropsychiatrist Ladislav Haškovec, who described the phenomenon in a non-medication induced presentation in 1901. [34] [1] Reports of medication-induced akathisia from chlorpromazine appeared in 1954. [a] Later in 1960 there were reports of akathisia in response to phenothiazines (a related drug). [1]