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Moeller's disease, Cheadle's disease, scorbutus, [1] Barlow's disease, hypoascorbemia, [1] vitamin C deficiency: Scorbutic gums, a symptom of scurvy. The triangle-shaped areas between the teeth show redness of the gums. Specialty: Endocrinology: Symptoms: Weakness, feeling tired, changes to hair, sore arms and legs, gum disease, easy bleeding ...
Certain vitamin and minerals deficiencies are common in people with porphyria cutanea tarda. The most common deficiencies are beta-Carotene, [7] retinol, [8] vitamin A [9] and vitamin C. Beta-Carotene is required to synthesize vitamin A and vitamin A is needed to synthesize retinol.
The historic importance of vitamin C deficiency relates to occurrence on long sea-going voyages, when the ship food supplies had no good source of the vitamin. Deficiency results in scurvy when plasma concentrations fall below 0.2 mg/dL, whereas the normal plasma concentration range is 0.4 to 1.5 mg/dL.
Vitamin B12 deficiency symptoms . Vitamin B12 deficiency has a few hallmark symptoms, according to doctors: Lack of energy. Mental fatigue. Nausea, vomiting, or diarrhea. Lack of appetite. Weight loss
Vitamin C (also known as ascorbic acid and ascorbate) is a water-soluble vitamin found in citrus and other fruits, berries and vegetables. It is also a generic prescription medication and in some countries is sold as a non-prescription dietary supplement. As a therapy, it is used to prevent and treat scurvy, a disease caused by vitamin C ...
This category reflects the organization of International Statistical Classification of Diseases and Related Health Problems, 10th Revision. Generally, diseases outlined within the ICD-10 codes E50-E56 within Chapter IV: Endocrine, nutritional and metabolic diseases should be included in this category.
Christoph Sator/picture alliance via Getty Images. A Catholic nun was arrested by Italian police on Thursday for bringing messages for the mafia to prisoners, police said in a news statement.
However, Jamaican pediatrician Cicely Williams introduced the term in 1935, two years after she published the disease's first formal description. Williams was the first to research kwashiorkor and differentiate it from other dietary deficiencies. She was the first to suggest that this might be a protein deficiency.