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Toxic encephalopathy is a neurologic disorder caused by exposure to neurotoxic organic solvents such as toluene, following exposure to heavy metals such as manganese, as a side effect of melarsoprol treatment for African trypanosomiasis, adverse effects to prescription drugs, or exposure to extreme concentrations of any natural toxin such as cyanotoxins found in shellfish or freshwater ...
The diagnosis is usually made by a brain scan , in which areas of swelling can be identified. The treatment for PRES is supportive: removal of the cause or causes and treatment of any of the complications, such as anticonvulsants for seizures. PRES may be complicated by intracranial hemorrhage, but this is relatively rare. The majority of ...
These seizures damage the frontal lobe's cognitive brain function such as memory and sensory abilities. This can result in learning disabilities, [6] behavioral disorders, memory issues, sensory changes, and possibly death. Children continue to have seizures throughout their lives.
Chronic solvent-induced encephalopathy (CSE) is a condition induced by long-term exposure to organic solvents, often—but not always—in the workplace, that lead to a wide variety of persisting sensorimotor polyneuropathies and neurobehavioral deficits even after solvent exposure has been removed.
A brain injury can cause seizure(s) because of the unusual amount of energy that is discharged across of the brain when the injury occurs and thereafter. A disruption of the supply of oxygen may cause damage to the temporal lobe of the brain. [35] The risk of seizure(s) from a closed head injury is about 15%. [36]
Occipital epilepsy can cause many seizures per day and often in multiple clusters. The seizures may also spread to other areas in the brain. Spreading of the seizures can move to the anterior regions, causing symptoms also from the frontal, temporal, and parietal lobes, and secondary hemi convulsions or convulsions. [5]