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Exercise-induced bronchoconstriction (EIB) occurs when the airways narrow as a result of exercise. This condition has been referred to as exercise-induced asthma (EIA); however, this term is no longer preferred. [1] While exercise does not cause asthma, it is frequently an asthma trigger. [1]
Other types of triggers like exercise, air pollutants, tobacco smoke, humidity, cold air, or certain medicines may also play a role in triggering asthma. [4] While it has been proposed that asthma triggers can be classified into three types: allergic triggers, environmental triggers and physical triggers, a universal categorization of asthma ...
More generally termed exercise-induced asthma, the preferred and more accurate term exercise-induced bronchoconstriction better reflects underlying pathophysiology.It is also preferred due to the former term giving the false impression that asthma is caused by exercise.
Exercise-induced asthma, technically called exercise-induced bronchoconstriction, or EIB, can make working out feel downright painful, if not altogether impossible. A narrowing of the airways that ...
Exercise can trigger bronchoconstriction both in people with or without asthma. [116] It occurs in most people with asthma and up to 20% of people without asthma. [116] Exercise-induced bronchoconstriction is common in professional athletes. The highest rates are among cyclists (up to 45%), swimmers, and cross-country skiers. [117]
Respiratory adaptation begins almost immediately after the initiation of the physical stress associated with exercise. This triggers signals from the motor cortex that stimulate the respiratory center of the brain stem, in conjunction with feedback from the proprioreceptors in the muscles and joints of the active limbs.
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In children, the most common triggers are viral illnesses such as those that cause the common cold. [22] Exercise or intense use of respiratory system—the effects of which differ somewhat from those of the other triggers, since they are brief. They are thought to be primarily in response to the exposure of the airway epithelium to cold, dry air.