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Although S. epidermidis is not usually pathogenic, patients with compromised immune systems are at risk of developing infection. These infections are generally hospital-acquired. [4] S. epidermidis is a particular concern for people with catheters or other surgical implants because it is known to form biofilms that grow on these devices. [5]
Potential probiotic treatment includes the use of Staphylococcus epidermidis to inhibit C. acnes growth. S. epidermidis produces succinic acid which has been shown to inhibit C. acnes growth. [42] Lactobacillus plantarum has also been shown to act as an anti-inflammatory and improve antimicrobial properties of the skin when applied topically ...
Starting very early, research into biofilm formation in Staphylococcus epidermidis has served as a model for other staphylococci such as Staphylococcus aureus and other CoNS species. Moreover, data also showed that S. capitis have a strain (AYP1020) that researchers use to general genomic characteristics compared to S. epidermidis’ strain ...
The S. haemolyticus strain JCSC1435 genome contains a 2,685,015 bp chromosome and three plasmids of 2,300 bp, 2,366 bp, and 8,180 bp. The chromosome is comparable in size to those of S. aureus and S. epidermidis and contains a similar G+C content. In addition, a large proportion of the open reading frames (ORFs) are conserved across all three ...
The formation of biofilm and structure of EPS share a lot of similarities with bacterial ones. The formation of biofilm starts with reversible absorption of floating cells to the surface. Followed by production of EPS, the adsorption will get irreversible. EPS will colonize the cells at the surface with hydrogen bonding.
The S. sciuri group appears to be the closest relations to the genus Macrococcus. S. pulvereri has been shown to be a junior synonym of S. vitulinus. [13] Within these clades, the S. haemolyticus and S. simulans groups appear to be related, as do the S. aureus and S. epidermidis groups. [14] S. lugdunensis appears to be related to the S ...
Dispersin B is produced by Aggregatibacter actinomycetemcomitans, a Gram-negative oral bacterium, when it needs to detach and disperse adherent bacterial cells. [4] A. actinomycetemcomitans forms asymmetric biofilm lobed colonies that release single cells or small clusters of bacterial cells, which can attach to nearby surfaces, form new colonies, and enable the biofilm to spread.
Staphylococcus hominis is a coagulase-negative member of the bacterial genus Staphylococcus, consisting of Gram-positive, spherical cells in clusters.It occurs very commonly as a harmless commensal on human and animal skin and is known for producing thioalcohol compounds that contribute to body odour.