Ads
related to: drugs that cause pseudotumor cerebri- Frequently Asked Question
Visit Our Website For Info
Regarding This Glioma Treatment.
- Possible Side Effects
Learn About the Possible
Side Effects of This Treatment
- Glioma & IDH1/2 Treatment
Do You Have An IDH1/2 Mutation?
Learn More About This Disease.
- Glioma Videos & Resources
Hear Rob's Story About Living With
Glioma & Staying Positive.
- Frequently Asked Question
mayoclinic.org has been visited by 100K+ users in the past month
Search results
Results From The WOW.Com Content Network
Idiopathic intracranial hypertension (IIH), previously known as pseudotumor cerebri and benign intracranial hypertension, is a condition characterized by increased intracranial pressure (pressure around the brain) without a detectable cause. [2] The main symptoms are headache, vision problems, ringing in the ears, and shoulder pain.
This is a list of drugs and substances that are known or suspected to cause Stevens–Johnson syndrome This is a dynamic list and may never be able to satisfy particular standards for completeness. You can help by adding missing items with reliable sources .
Oral: Differentiation syndrome, hyperleucocytosis, elevated cholesterol and triglycerides, arrhythmias, pancreatitis, elevated liver enzymes, thrombosis, intracranial hypertension and pseudotumour cerebri (mainly in children), anxiety, depression and genital ulceration (rare). Topical: Erythema. 2.4 Immunomodulatory Agents (IMiDs) Lenalidomide: PO
Brain tumor, idiopathic intracranial hypertension (also known as Pseudotumor Cerebri), cerebral venous sinus thrombosis or intracerebral hemorrhage; Respiratory failure [4] Isotretinoin, which is a powerful derivative of vitamin A, rarely causes papilledema. medications like tetracycline
Drug-induced intracranial hypertension (DIIH) or medication-induced intracranial hypertension is a condition of higher than normal intracranial pressure with the main cause being a drug. [15] This condition is similar to idiopathic intracranial hypertension , however the etiology in this instance is a drug. [ 16 ]
Management of cranial venous outflow obstruction involves treating the underlying cause, if identifiable, and managing the symptoms. This can include medication to reduce intracranial pressure, anticoagulation therapy to prevent thrombosis, and in some cases, surgical intervention to restore normal venous drainage. [6] [11]
Minocycline has also been reported to very rarely cause idiopathic intracranial hypertension (pseudotumor cerebri), [48] a side effect also more common in female patients, potentially leading to permanent vision damage if not recognized early and treated. [49]
Any medications within the family of antipsychotics can cause the condition, though typical antipsychotics appear to have a higher risk than atypicals, [1] specifically first generation antipsychotics like haloperidol. [5] Onset is often within a few weeks of starting the medication but can occur at any time.