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The three different mechanisms of high iodine response, the Plummer effect, the Wolff-Chaikoff inhibition effect, and the adaptive escape phenomenon, synergistically work together to fend off potentially harmful consequences of excess iodine load and ensure thyroid homeostasis. [1]
Wrong timing of iodine use may even increase the risk by triggering the Plummer effect. [16] The Plummer effect, the Wolff-Chaikoff inhibition effect, and the adaptive escape phenomenon, synergistically work together to fend off potentially harmful consequences of excess iodine load and ensure thyroid homeostasis. [17]
One study concluded the range of total body iodine content in males was 12.1 mg to 25.3 mg, with a mean of 14.6 mg. [77] It is presumed that once thyroid-stimulating hormone is suppressed, the body simply eliminates excess iodine, and as a result, long-term supplementation with high doses of iodine has no additional effect once the body is ...
This is an accepted version of this page This is the latest accepted revision, reviewed on 10 February 2025. This article is about the chemical element. For other uses, see Iodine (disambiguation). Chemical element with atomic number 53 (I) Iodine, 53 I Iodine Pronunciation / ˈ aɪ ə d aɪ n, - d ɪ n, - d iː n / (EYE -ə-dyne, -din, -deen) Appearance lustrous metallic gray solid ...
Iodine plays a role in thyroid production, and excess iodine levels within the body can result in overproduction of thyroid hormone. Initially, the thyroid reacts according to the auto-regulatory Wolff-Chaikoff effect to prevent an excess of thyroid hormone production. [5] Usually, the thyroid normalizes within 24-48 hours.
Too much sodium can raise blood pressure, and the risk for heart disease and stroke, the agency warns. The FDA has been urging the food industry to use less salt, noting that could prevent ...
However, unlike the Plummer and Wolff-Chaikoff effects, the Jod-Basedow effect does not occur in persons with normal thyroid glands, as thyroid hormone synthesis and release in normal persons is controlled by pituitary TSH secretion, which does not allow hyperthyroidism when extra iodine is ingested. [citation needed]
Excessive iodine intake is a well-established environmental factor for triggering thyroid autoimmunity. Thyroid autoantibodies are found to be more prevalent in geographical areas with a higher dietary iodine levels. Several mechanisms by which iodine may promote thyroid autoimmunity have been proposed: